Includesa bibliographical references and index.
|Statement||G. Bruschi, A. Borghetti (eds.)|
|Contributions||Bruschi, G. 1946-, Borghetti, Alberico.|
|LC Classifications||RC685.H8 C378 1991|
|The Physical Object|
|Pagination||xii, 271 p. :|
|Number of Pages||271|
|ISBN 10||3540539875, 0387539875|
|LC Control Number||91004839|
Offers the comprehensive views of specialists in cellular research in hypertension. Converging work in the fields of clinical medicine and cellular biology is also covered in detail and topics include receptor occupancy, transmembrane signalling and intracellular messengers. hypertension or high blood pressure, elevated blood pressure resulting from an increase in the amount of blood pumped by the heart or from increased resistance to the flow of blood through the small arterial blood vessels (arterioles). Hypertension was generally defined as a blood pressure reading of over 90 or higher, but new guidelines issued in define hypertension as over 80 or. Hypertension, usually defined as persistent blood pressure at /90mmHg or higher, affects about a quarter of the adult population in many countries and particularly in Western societies. Hypertension is a risk factor for most, if not all, cardiovascular diseases and renal failure. While blood pressure should be measured repeatedly for the diagnosis, new techniques such as hour ambulatory Author: Sverre E. Kjeldsen. Cellular and Molecular Biology of the Renin-Angiotensin System provides the first review and update of the state-of-the-art cellular and molecular aspects of the renin-angiotensin system. The book presents detailed analyses from world experts on each component of this system, including future directions. Topics range from angiotensin II receptor subtypes to processing of renin Cited by:
A unique aspect of the book is that it covers the effects of exercise mimetics on vascular cell adaptations in order to begin to elucidate some of the cellular mechanisms that may underlie blood pressure reductions with exercise training. Lastly, the book will end with a chapter on the interactive effects of genes and exercise on blood pressure. The aim of the present work was to use LSCM to study in intact resistance arteries the cellular changes associated with remodeling in hypertension. We first assessed the ability of the method to obtain simple dimensional measurements compared with the two methods presently available: wire Cited by: Book review: Hypertension, 2nd edition March Br J Cardiol ; Leave a comment. particularly involving the epidemiological and pathophysiological aspects of the subject. Whilst this detracts from reading the book as a continuous text, it does facilitate its use as a handy reference. The British Journal of Cardiology : Terry Mccormack. Hypertension: pathophysiology and treatment. Environmental Aspects of Hypertension. Chapter. Full-text available. cellular adhesion molecules and cytokines. An alternative approach.
Diabetes, Hypertension and Cardiovascular Disease-An Unsolved Enigma. Hypertension and Cardiovascular Disease-An. Various aspects of these two prevalent diseases among the places : Maitree Bhattacharyya. A unique aspect of the book is that it covers the effects of exercise mimetics on vascular cell adaptations in order to begin to elucidate some of the cellular mechanisms that may underlie blood pressure reductions with exercise training. Lastly, the book will end with a chapter on the interactive effects of genes and exercise on blood : $ Genetic and Molecular Aspects of Hypertension. Sandosh Padmanabhan, Mark Caulfield, and ; Anna F. Dominiczak; From the Institute of Cardiovascular and Medical Sciences, College of Medical, Veterinary and Life Sciences University of Glasgow, Glasgow, United Kingdom (S.P., A.F.D.); and Queen Mary University of London, Barts and The London School of Medicine, Clinical Pharmacology, London, Cited by: Hypertension with an identified cause (secondary hypertension) is usually due to sleep apnea, chronic kidney disease, or primary aldosteronism. Usually, no symptoms develop unless hypertension is severe or long-standing. Diagnosis is by sphygmomanometry. Tests may be done to determine cause, assess damage, and identify other cardiovascular risk.